How Does Chelation Therapy Work?
Does EDTA Have Side Effects?
When Is Chelation Therapy Needed?
And How About The Scientific Proof?
Chelation Therapy In Practice
EDTA chelation therapy is a form of treatment aimed at reducing calcium deposits, removing the heavy metals that inhibit enzyme systems, controlling lipid peroxidation, & reducing platelet “stickiness” in the clinical management of atherosclerosis & related disorders. EDTA chelation therapy is also used in the treatment of heavy metal intoxications, hypercalcemia, (high calcium in the body) & the control of ventricular arrhythmias secondary to digitalis intoxication. (Remedy used for the heart).
EDTA chelation therapy also reduces calcium deposits in cells & arteries. The best results were achieved in treatment & prevention of blocked arteries in the brain (prevention of hemi-plegia, memory loss & other problems), in the coronary heart vessels (helps avoiding Bi-pass surgery), in the legs (helps preventing leg amputations, restless-leg syndrome, pain in the legs & others). Each session last 60-120 min.
Important scientific discoveries often occur coincidentally. The history behind the development of chelation therapy certainly confirms this fact. In the Thirties, EDTA, the active component in chelation therapy, was discovered by a German, Munz. He had been searching for a substance which could soften hard water, and it appeared EDTA was highly effective in doing so. But EDTA had the capacity to do much more. The substance happened to have a strong affinity for (toxic) heavy metals such as lead, mercury and cadmium, and actually binds with them.
For this reason, EDTA was initially used in treating acute lead intoxication. Lead, an extremely toxic substance to the human body, was bound to EDTA injected into the blood stream and thus simply removed from the body during urination. With EDTA, however, fate became the mother of invention.
Patients suffering from cardiovascular disease, who where being treated with EDTA for acute lead intoxication, noticed improved stress tolerance and less chest pain upon exertion. They were also able to walk longer distances. Surprisingly, chelation therapy appeared to have produced an unexpected healing effect for their cardiovascular problems.
This finding led to further investigation. It was Norman Clarke who, in 1956, published an article in The American Journal of Medical Science, claiming the successful treatment of patients with severe angina pectoris (chest pain caused by narrowing of the coronary arteries) through the use of EDTA. In 1960, well-known cardiologists Kitchell and Meltzer reported that chelation therapy caused both objective and subjective improvement in 9 out of 10 patients suffering from angina.
Soon hereafter, numerous publications on the successful treatment of patients with narrowed leg or brain arteries started appearing.
In 1988, Olszewer and Carter published their findings after treating 2870 patients with EDTA chelation therapy. More than 93% of the patients suffering from narrowed coronary arteries, showed good to excellent improvement. Patients suffering from the narrowing of leg arteries improved in 97% of all cases. And patients suffering from narrowed brain arteries also profited, although less dramatically (60%).
All in all, these results have led to in increased interest in the effectiveness of chelation therapy. The more than 10 million treatments administered to date are proof of this effectiveness.
EDTA (short for ethylenediaminetetraacetic acid) is the active component in chelation therapy and is a chelator or chelating substance. This term stems from the Greek word "chele", meaning claw, as EDTA actually grabs on to metal particles (such as calcium, lead, mercury, cadmium) like a claw. Once injected into the blood stream, it binds to these particles and this entire complex (EDTA plus metal particles) is excreted from the body during urination. The positive effects of chelation therapy can be seen in the following:
It is the process of arteriosclerosis which causes narrowing of the arteries of heart, legs and brain. Here, calcium deposits (plaque) are found in the arterial wall. EDTA binds calcium ions (electrically charged calcium particles) in the blood and excretes them via the kidneys into the urine. Thus, the blood calcium ion concentration will drop and (in response to this) will lead to the release of parathormone (PTH). PTH in its turn will attract calcium ions from the tissues (including the arterial walls) in order to compensate for the lowered blood calcium concentration. Were this PTH reaction not to happen, the blood calcium concentration would drop still further and lead to involuntary contractions of numerous muscles (tetany).
Upon ageing, the calcium concentration of body cells will gradually increase. This leads to impairment of metabolic processes and energy production within the cell (in other words: impaired cell function). Chelation therapy will decalcify these cells, thereby improving their function.
One of the most important functions of EDTA is the removal of heavy metals from the body. Upon ageing, substances such as lead, mercury and cadmium accumulate in the body ("internal pollution"). Present day lead load in humans, for instance, is about 1000 times higher than 1600 years ago. Heavy metals exert very damaging effects upon the body, amongst other things because they block numerous enzymes (substances which accelerate certain bodily processes).
EDTA also removes iron and copper from the body. These metals are involved in so-called free radical reactions which take place in the body. Free radicals are atoms or molecules (containing a unpaired electron) which tend to interfere with numerous bodily processes in an extremely aggressive fashion. For instance, free radicals can bind to enzymes, cellular membranes or DNA (the genetic material) and thus interfere with their function. Given the proper conditions, free circulating iron and copper ions can promote the formation of free radicals. Chelation therapy will remove an excess of these ions from the body and thus exert a positive effect on these damaging reactions. This EDTA effect explains the positive influence on arthritic complaints which, in part, are caused by free radicals.
EDTA has a positive effect on blood platelets (thrombocytes). These play an important role in cardiovascular and other arterial disease. Generally, brain and myocardial infarctions occur because clots (which are largely made up of platelets) close off a vessel.
Moreover, blood coagulation tendency in arteriosclerotic disease appears to be greater than normal, thus facilitating the formation of clots. EDTA, however, reduces platelet stickiness, thereby considerably reducing the chance of clot formation.
EDTA influences red blood cells
Red blood cells (erythrocytes) contain oxygen, the essential component every cell needs. Through chelation, red blood cells become increasingly flexible, making them better capable of reaching smaller blood vessels and thus increasing circulation and oxygen delivery.
Angina pectoris: chest pain as a consequence of lack of oxygen of the heart muscle. This pain generally occurs upon exertion, in cold weather, emotional states or upon eating copious meals, but sometimes also when in a resting state and at night.
Heart attack (myocardial infarction): here, part of the heart muscle has actually died off, generally as a consequence of an acute blockage of a coronary artery. Chelation therapy helps prevent new heart attacks, as well as angina pectoris after a heart attack. Moreover, chelation improves cardiac pump function.
Prevention of bypass surgery or balloon angioplasty: large American and European studies show that only a small percentage of patients undergoing such procedures will experience long term effects, as compared to patients treated with medication only.
The initial stage of this condition is called intermittent claudication, where patients suffer from different degrees of pain. Gangrene (actual tissue die-off as a consequence of blockage or severe narrowing of a blood vessel) can be the next stage (the so-called "black toe" is a good example of this).
These can manifest themselves in the following ways:
Cerebral infarction: die-off of brain tissue as a consequence of blockage of a blood vessel by clot build-up onto an already narrowed segment (thrombosis), or blockage caused by a loosely circulating clot which became lodged (embolism).
TIA (Transient Ischaemic Attaque): temporary lack of oxygen of the brain, which completely reverses itself within 24 hours (such as transient speech difficulties, transient paralysis, transient blindness in one eye, etc.).
Memory and concentration difficulties
Impaired vision and hearing
Fatigue, decreased vitality
The majority of patients experience an increase in energy and vitality as a consequence of chelation therapy.
Here, chelation therapy is also frequently effective. However, additional measures are almost always necessary.
Chelation therapy improves sugar metabolism. Diabetics undergoing chelation therapy often need less insulin or oral blood glucose lowering medication during the course of treatment. Moreover, chelation therapy helps prevent complications of diabetes, such as eye, nerve and kidney damage, as well as accelerated arteriosclerosis.
High blood pressure (hypertension)
Chelation therapy will generally lower raised blood pressure. Blood pressure lowering medication can frequently be diminished or even discontinued.
Prevention, "life extension" - striving for optimum health
Because of the favourable effects of chelation therapy on free radicals, heavy metals and calcium deposits, degenerative processes (arthritis, general "wear-and-tear") of the body will be slowed down. Not only will the body's blood vessels be kept in good condition, but the entire body benefits from regular EDTA infusions. Research in laboratory animals shows a clear increase in life expectancy through EDTA. In humans, increase in life expectancy is estimated at 8-17 years in men and 6-16 years in women (an average of 12 years). Life extension in and by itself would not make sense if the quality of life were not equally effected in a positive fashion. Chelation therapy clearly has this effect as well.
The argument many conventional doctors use in their criticism of chelation therapy is that the effects have never been properly proven. Here follows a summary of some trials on the effects of chelation therapy. This is only the tip of the iceberg when it comes to the amount of available trials:
Are you interested in chelation therapy? The first step would be for you to set up an intake appointment. Not only will your current complaints be analysed, but also other matters such as your medical history, life-style, use of medication etc. will be addressed. Afterwards, a physical examination is performed to assess your condition more accurately. If you are only interested in an informative consultation (where further examinations may not be necessary), the doctor will advice you, based on the medical data you present.
In addition to the basic physical examination, the doctor will often need extra information. Quite regularly, an electrocardiogram (EKG) and a bicycle stress test will be needed to assess the condition of the heart and blood vessels. Laboratory blood tests will be needed to assess possible liver or kidney function disturbances, raised blood lipids (cholesterol), infections, etc. Sometimes, a pulmonary function test is necessary.
If you are being, or have been, treated by other medical doctors or specialists for your condition, you may be asked for approval to have your specialist supply additional medical data. This is to avoid duplication of tests and to better inform the doctor about your condition.
After compiling all the necessary data, the results will be discussed with you. Based on these, a decision is made as to whether you are eligible for chelation therapy or whether other therapies are more appropriate.
As mentioned earlier, chelation therapy is applied via a drip. A special needle is inserted into one of the veins in the arm. Once in the vein, the actual needle is removed, leaving a small, flexible plastic catheter in place. This catheter allows for a certain amount of mobility; you may bend your arm, walk around, go to the toilet, etc. After hooking up the drip bottle to the catheter, the appropriate infusion rate is set. For optimal effect, the average length of a chelation session is 1.5 to 3 hours.
The treatment regime for a diagnosed disease or condition consists of a series of 20-40 drips. These are given twice a week. After this series, the frequency is gradually reduced to one monthly maintenance session. This is done in order to prevent the possible further progress of the treated condition.
Individual adjustments of treatment frequency and intervals is possible, but may not always be advisable. The maximum effect of chelation is generally not reached until 2 to 3 months after the 20th drip.
This does not imply that improvement of complaints will not become visible much faster. Some patients report clear progress after 5 drips, others will not experience anything until the 15th bottle.
The use of chelation therapy as a form of prevention does not require such a large number of sessions. A series of 10 drips, administered twice per week, tends to be the norm. The necessity or frequency of maintenance sessions can be decided upon on an individual basis.
If you are a smoker and want to start chelation therapy, you should quit. Smoking impairs the effectiveness of chelation therapy and may show disappointing results. Nicotine patches, acupuncture or hypnotherapy are only some of the ways to help you kick the habit.
A proper diet is also important. In short, this means limiting the intake of saturated (mainly animal) fats, as well as, the use of refined products and sugar. Furthermore, your diet will need to be supported with nutritional supplements: mega-dose vitamins, minerals, trace elements, (co)enzymes, etc. These may well have a positive influence on the disease process and are vital in preventing shortages of certain vitamins and minerals which chelation therapy tends to deplete.
Intravenous chelation therapy for heavy metal De-tox, mercury in particular, with vitamin C (50 grams), Glutathione & some times Tourine.
Vitamin C will improve your immune system, prevents the likelihood of infection after surgeries, will give you energy & other benefits
Glutathione enhances the elimination of heavy metals, also good for the musculo-skeletal system & neurological disorders, chronic fatigue syndrome & will give you energy. It is produced naturally in the body as part of Krebs cycle.
Tourine enhances the elimination of heavy metals from the brain & the heart.
Although we use the best techniques for safe Amalgam removals, we recommend IVC to be given during the same period of Amalgam removals thus eliminating any risks of mercury toxicity from the fumes generated from Amalgam removal.
Heavy metals are eliminated through the colon. Each session lasts between 60-90 min.
This chelating agent is the strongest & the best mainly used for mercury toxicity. DMPS also chelates mainly lead, cadmium, silver, tin, arsenic, copper, zinc & in-organo-mercury compounds.
DMPS should be used only after the removal of all mercury-silver amalgam fillings.
Post-DMPS mineral solution is given after finishing the course of treatment.
DMPS should be given once every 2-4 weeks. Heavy metals are eliminated through the kidneys. Each session lasts about 30-60 min.
With chelation therapy, at all times, mineral & vitamin supplements are given intravenously & orally during or after the session depending on the protocol. Special dietary recommendations advised.
Blood, Hair & Saliva Analysis Test for Candida antibody, Heavy metal sensitivity, Serum mineral & vitamins screening, & others.
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